Answer: The pathology described in the question is "testicular torsion".
The most sensitive physical exam that is specific to this pathology is testing for reflexes. The inner thigh of the affected side when rubbed should cause the testicle to contract; but in this pathology, it does not.
Explanation:
Testicular torsion is a condition caused by the twisting of the spermatic cord, which causes a loss of blood flow to the testicle. It is the leading cause of testicular loss in adolescent boys, if no surgical intervenvention is carried out as soon as possible.
Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
<span>Enzymes
These are characterized by macromolecular natural impetuous with catalysts that quicken synthetic responses. The particles where upon proteins may act are called substrates and the chemical changes over the substrates into various atoms which are their by products</span>
Answer:
Glycerophosphipids form the lipid bilayer
The answer is D because if its dry outside you will have extreme drought
