<span><span>This
is because his hypothesis that an organism can pass on characteristics that it
has acquired during its lifetime to its offspring are not approved or </span>verified.
His hypothesis of the notion of inheritance of acquired traits was supplanted
by Mendelian genetics. Yet, because of the discovery of epigenetics and somatic
hypermutation interest in Lamarckism has continued.</span>
Cyclins
are the chemicals that regulate the cell cycle. Cyclins work by regulating the timing
of the cell cycle in eukaryotic cell. Cyclins activates cyclin dependent
kinases (CDKs) (an enzyme that works by adding <span>negatively charged phosphate groups to other
molecules in a process called phosphorylation) by binding to it to form a cyclin-Cdk
complex. This complex then functions by acting as a signal to the cell to move
to the next cell cycle phase. At the end of the event, the cyclin is degraded, Cdk
is deactivated, therefore signaling exit from a specific phase.</span>
Answer:
Malignant melanoma cells would have active telomerases that constantly replenish and lengthen telomeres.
Explanation:
Telomerase can be described as enzymes which add the repetitive sequences called telomeres at the end of a chromosome. Telomeres can be described as repetitive sequences at the end of the chromosome which are involved in protecting the chromosome from any damage.
In a normal skin cell, the telomeres will shorten with time. But in a malignant skin cell, the telomerase will add the repetitive sequence again and again. The telomers will not be able to shorten.
Answer:
This protein doesn't have a secondary structure (i.e., alpha helices or beta sheets), thereby it interacts with the specific molecule trough its loops
Explanation:
Protein loops may interact with diverse molecules including nucleic acids (RNA and DNA), proteins, signal molecules, etc. These protein sequences (loops) interact with both polar and non-polar head groups
