Answer:
The type of vesicular transport involved in the exporting of protein-based hormones, such as insulin, into the bloodstream is regulated exocytosis.
Explanation:
In general terms, exocytosis is a type of active transport that allows intracellular substances are released to the extracellular space, through of vesicles that fuse with the plasma membrane, which allow the exit of substances from inside the cell.
Regulated exocytosis is the specific vesicular transport for the secretion of substances, such as hormones. For this type of transport to exist, the presence of an extracellular signal is required, which will activate the fusion of the vesicles.
In the case of insulin, the external signal originates with the increase in blood glucose levels, a signal that penetrates the intracellular space and generates an increase in insulin production in the islets of Langerhans (pancreas).
Before insulin secretion occurs, the cell must be depolarized, allowing calcium to enter, which promotes transport by regulated exocytosis of insulin to the extracellular space.
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brainly.com/question/1109181
In Pavlov's original studies on classical conditioning, he used dogs as the subject of his experiment. The unconditioned stimulus was the <u>bowl of food</u> that he presented to the dog while the unconditioned response was the dog salivated in its excitement to see the food.
Answer:
When the level of calcium ions declines the sarcoplasm stops the sliding.
Explanation:
Sarcoplasm can be described as the cytoplasm for the cells of muscles. It contains important structures such as the myofibrils.
The following steps would occur when the level of calcium ions would decline:
The sarcoplasm would stop the sliding of the thin filaments.
The actin filaments would be migrated in the M line
This would lead the bands in the I region to shorten.
The Z region would compress.
The H region would diminish.
Mycelium-Fungi
<span>
Heterotrophic-Both</span>
<span>
Pseudopods-Protists</span>
<span>
Contain cell walls with chitin-Fungi </span>
<span>
Fruiting body-Fungi</span>
<span>
Flagellum-Protists
I hope this helps out alot. </span>
Answer:
Explanation:
NADH and FADH2 are both electron carriers of the electron transport chain. NADH gives up its electrons starting from Complex I, which has a higher energy level compared to other complexes. Energy is given off to pump protons across the membrane by the time electrons are transferred to ComplexIII. More electrons are pumped across the membrane as electrons move to Complex IV. Because NADH commenced giving up its electrons from Complex I (higher energy level complex), more protons are pumped across the membrane gradient, which enables ATP synthase with more power to produce 3ATP molecules per NADH molecule.
On the other hand, 2 molecules of ATP are generated by FADH2 because it starts by giving up its electrons to ComplexII. It missed a chance to pump protons across the membrane when it passed Complex I. By the time the electrons reach Complex IV, less protons have been pumped. The lesser the protons to power ATP synthase, the lesser the ATP molecules produced.
