Answer:
A. 1' ... 5'
Explanation:
The pentose sugars present in nucleotides have five-carbon structures. Other components of a nucleotide are a phosphate group and a nitrogenous base. The nitrogenous base in a nucleotide can be a purine or pyrimidine. The purine bases are covalently joined to the 1' carbon atom of the pentose sugar at their N-9. The bond between the nitrogenous base and a pentose sugar is called the N-beta glycosyl bond.
Similarly, N-1 of a pyrimidine base is covalently linked to the 1' carbon of the pentose sugar. The phosphate group of a nucleotide is bonded to the 5' carbon of the pentose sugar.
Answer:
High-energy phosphorylated intermediates that serve as phosphate donors to ADP are generated as a result of electron-transfer reactions.
Explanation:
Peter Mitchell proposed Chemiosmotic hypothesis which state that the driving of the synthesis of ATP is accomplished by a proton-motive force. He propose that whenever electron is moved along the electron transfer chain then proton will be moved accross the inner part of mitochondria membrane. As a result of this there would be increase in PH of the mitochondria matrix and reduced PH of the intermembrane space.
Some Evidence about Chemiosmotic hypothesis hypothesis;
1)transportation of electron brings about the generatation of a proton gradient. there is difference in the PH on the outside and inside the mitochondria.
2)To synthesis ATP, a proton gradient is required only and other means by which PH gradient is generated
Therefore,High-energy phosphorylated intermediates that serve as phosphate donors to ADP are generated as a result of electron-transfer reactions is not Micheal Mitchell statement.
I would say the main term is myocardial infarction and the modifier is acute which means that it is serious but a discrete one time thing as opposed to a chronic condition like say arthritis that is usually with a person for many successive years rather than just occurring once.
Answer:
The correct answer is C
Explanation:
I took the test on edg2020
Answer: option A, B and D
Explanation:
The telomeres found at the terminal ends of eukaryotic chromosomes are long repetitive sequences that protects the DNA from being identify as a damage molecule to encourage fusion with other chromosomes. Since replication does not extend down to these 3' ends, the cell recruits the telomerase enzyme which is a ribonucleoprotein that helps to add telomeric repeats using its own RNA component as template to extend this end. Thus if mutation like a large deletion occurs in the gene encoding for this enzyme, the telomerase would not be able to associate correctly with the telomere, the catalytic center also encoding for its RNA polymerase would be altered and the enzymes will not be able to synthesize new telomeric repeats sequences
